title: Molecular mathematical model of the skeletal muscle reduction
metadata.name: dc.contributor.author
metadata.value: Novoselov, Viktor S.
metadata.lang: -
metadata.name: dc.date.accessioned
metadata.value: 2016-11-24T19:12:42Z
metadata.lang: -
metadata.name: dc.date.available
metadata.value: 2016-11-24T19:12:42Z
metadata.lang: -
metadata.name: dc.date.issued
metadata.value: 2016-09
metadata.lang: -
metadata.name: dc.identifier.citation
metadata.value: Novoselov V. S. Molecular mathematical model of the skeletal muscle reduction. Vestnik of Saint Petersburg University. Series 10. Applied mathematics. Computer science. Control processes, 2016, issue 3, pp. 88–96.
metadata.lang: en_GB
metadata.name: dc.identifier.other
metadata.value: 10.21638/11701/spbu10.2016.308
metadata.lang: -
metadata.name: dc.identifier.uri
metadata.value: http://hdl.handle.net/11701/5693
metadata.lang: -
metadata.name: dc.description.abstract
metadata.value: All muscle contraction occurs as a result of signals in the form of the nervous impulses. The
  muscle fiber tension generales at first isometric and next isotonic contractions. In isometric
  contraction the muscle remains the same length. In isotonic contraction the tension in the muscle
  remains constant. In reality more complex contractions (isovelocity, concentric and eccentric)
  occur. However the cancelable mechanism will be identical. In the sliding filament theory it is
  a process of repetitive events that cause a thin filament (protein actin) to slide over a thick
  filament (protein myosin) and generate tension in the muscle. Briefly the mechanism of muscle
  contraction is as follows. The ATP molecules to activate the myosin head on the thick filament.
  An action potential arrives across the neuromuscular synapse to the muscle membrane. This
  depolarization results in an increase in cytoplasm calcium. This calcium to release calcium
  out the sarcoplasmic reticulum. The calcium ions to activate the binding cites on the thin
  filament. As a result of the ferment reaction take place hydrolyzes the ATP and the release of
  inorganic phosphate. In the article the standard problem of the sarcomere in the cause of the
  length-tension curve is constructed. The equilibrium solutions of the kinetic equations for the
  two models of muscle contraction are obtained. The mitochondrial phosphorylation of ADP
  is a complex process of cellular respiration. In this article only one fragment for the electron
  transport chain is considered. Refs 11.
metadata.lang: en_GB
metadata.name: dc.language.iso
metadata.value: ru
metadata.lang: en_GB
metadata.name: dc.publisher
metadata.value: St Petersburg State University
metadata.lang: en_GB
metadata.name: dc.relation.ispartofseries
metadata.value: Vestnik of Saint Petersburg University. Series 10. Applied Mathematics. Computer Science. Control Processes;Issue 3
metadata.lang: -
metadata.name: dc.subject
metadata.value: skeletal muscle
metadata.lang: en_GB
metadata.name: dc.subject
metadata.value: muscle contraction
metadata.lang: en_GB
metadata.name: dc.subject
metadata.value: molecular mathematical model
metadata.lang: en_GB
metadata.name: dc.subject
metadata.value: kinetic equations
metadata.lang: en_GB
metadata.name: dc.subject
metadata.value: mitochondrial phospholation
metadata.lang: en_GB
metadata.name: dc.title
metadata.value: Molecular mathematical model of the skeletal muscle reduction
metadata.lang: en_GB
metadata.name: dc.type
metadata.value: Article
metadata.lang: en_GB
resources.filename: 08-Novoselov.pdf
resources.url: https://dspace.spbu.ru/bitstream/11701/5693/1/08-Novoselov.pdf
resources.format: Adobe PDF
resources.size: 215,29 kB